Professional Education
Evidence-based materials for clinicians involved in the diagnosis, prevention and management of peri-implant diseases.
This section provides educational materials for dental professionals, including clinicians, periodontologists, implantologists and other specialists involved in implant dentistry. The content focuses on the clinical, biological and diagnostic aspects of peri-implantitis, with particular attention to risk factors, disease progression, inflammatory mechanisms, radiological assessment and long-term implant maintenance. The materials are designed to support evidence-based clinical thinking and to help professionals better understand peri-implant tissue destruction as a multifactorial process involving microbial, host-related, biomechanical and material-related factors.
Aggregated inflammatory indices in peri-implantitis
Aggregated inflammatory indices, such as NLR, PLR, MLR, SII, SIRI, and AISI/PIV, are attracting increasing attention because they allow inflammation to be viewed not through a single marker, but through the relationships between different elements of the immune response. In peri-implantitis, this may be particularly relevant, as the disease develops
From CRP to SII. Why may single inflammatory markers not be enough?
CRP, leukocytes, and pro-inflammatory cytokines are among the most recognizable markers of inflammation. In the context of peri-implantitis, they may provide valuable information about the activity of the inflammatory response, but they also have important limitations. A single marker reflects only one fragment of a complex biological process and usually
Can a blood test help assess a patient with peri-implantitis?
Peri-implantitis is diagnosed primarily on the basis of clinical and radiological assessment: evaluation of peri-implant tissues, bleeding on probing, probing depth, suppuration, implant mobility, and bone loss. Increasingly, however, the question arises whether in some patients it is worth looking more broadly — also at the systemic inflammatory response. Blood
The implant patient as a systemic patient. How do chronic diseases affect the risk of peri-implantitis?
Peri-implantitis is becoming increasingly difficult to describe solely as a local complication around an implant. Bacterial biofilm remains a key factor initiating inflammation, but the course of the disease may be modified by the patient’s systemic health, metabolism, chronic diseases, and immune response profile. Diabetes, obesity, metabolic syndrome, cardiovascular disease,
Why can peri-implantitis progress faster than periodontitis?
Peri-implantitis is often informally compared to periodontal disease developing around an implant. This comparison is understandable, but incomplete. Although periodontitis and peri-implantitis share the involvement of bacterial biofilm and a chronic host inflammatory response, the tissues surrounding an implant differ from those around a natural tooth. Different anatomy, altered soft
Peri-implantitis does not end with biofilm
For many years, peri-implantitis has been described primarily as a consequence of bacterial biofilm and the local inflammatory response around the implant. This model remains fundamental to understanding the disease, but it is becoming increasingly clear that it does not fully explain the complexity of the problem. Bone loss around